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Postday Weight loss clinics in phoenix phentermine levonorgestrel 0.75 mg (n = 11) placebo 0.75 mg (n = 11) Phentermine pills for cheap levonorgestrel 2.5 mg (n = 11) levonorgestrel 3.5 mg (n = 11) n 11 12 wk = 13 12-wk n 14 wk = Baseline Week 24 36 Dose of study drug Change from baseline to endpoint Week 24 36 Dose of study drug Change from baseline to endpoint Week 24 36 Dose of study drug Change from baseline to endpoint View Large FIGURE 2. View largeDownload slide Flow chart of selected trial interventions. Statistical analyses. Baseline values are summarized as mean ± SD and are shown as box plots of means and percentages across studies. The statistical threshold for significance was set at P < 0.05. Analyses were carried out with SAS (SAS Institute), version 9.1 Institute). Descriptive statistics for data from the 13 studies are shown in TABLE 2. Fluoxetine did not significantly affect the number of orgasms that resulted from oral contraception (difference: 0.12 ± 0.14 vs 0.06 0.09, and −0.23 ± 0.19 vs 0.08 0.12). In contrast, norethindrone did increase the number of orgasms experienced by women taking daily norethindrone 1.3 ± 0.25 compared with 0.1 0.07, P = 0.046. An increase in orgasms occurred for women taking levonorgestrel 0.75 mg (0.15 Xanax 1mg 60 $235.00 $3.92 $211.50 ± 0.19 vs −0.05 0.10; and 0.31 ± 0.19 vs 0.06 0.16 [see eTable 1 in the Supplement and eFigure 3 in the Supplement], P = 0.003) and levonorgestrel 2.5 mg (0.10 ± 0.08 vs 0.11 0.12 [see eTable 2 in the Supplement], P = 0.05) on the same day. An increase in orgasm was not observed for women taking levonorgestrel 3.5 mg daily (n = 11 [difference: 0.11 ± 0.07 vs 0.09, P = 0.96]; 0.96) or luteal-phase progesterone 0.5 microg (0.09 ± 0.04 vs 0.05 0.01, P = 0.47). Women taking norethindrone, levonorgestrel, or luteal-phase progesterone, but not the three other progestational hormone preparations tested (luteal-phase progesterone and the other progestagen, progesterone, ethinyl estradiol), differed significantly from those taking no progestational hormones on the primary variable of sexual desire, as determined by the BDI scale (difference: 0.13 ± 0.08 vs −0.24 0.25, P = 0.002; < 0.001 and P 0.02 for luteal-phase progesterone, norethindrone, and the other progestagen, progesterone, ethinyl estradiol, respectively), which reflects a woman's desire for intercourse. Compared with women taking levonorgestrel, an increase in sexual desire was observed only if they were taking levonorgestrel 2.5 mg (difference: 0.02 ± 0.08 vs 0.04 0.02, P = 0.22 and < 0.05 for levonorgestrel, luteal-phase progesterone, ethinyl estradiol, and norethindrone, respectively), a difference that was driven by women taking luteal-phase progesterone 5 microg, norethindrone, or levonorgestrel 3.5 mg (P < 0.001 and P 0.02, respectively). For women taking norethindrone or the other progestogenic hormone preparations, there was no difference between groups for sexual desire as assessed on the Sexual Desire Inventory. However, women taking luteal-phase progesterone 0.5 microg (P < 0.1 and P = 0.6 for levonorgestrel, luteal-phase progesterone 0.5 microg [difference: −0.01 ± 0.02 vs 0.03 0.02], P = 0.8], luteal and luteal-phase progesterone 5 microg, norethindrone, or levonorgestrel 3.
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